Objects
Schuliga, Michael, Pechkovsky, Dmitri V., Mutsaers, Steven E., Jaffar, Jade, Westall, Glen, Grainge, Christopher, Knight, Darryl A., Read, Jane, Waters, David W., Blokland, Kaj E. C., Reid, Andrew T., Hogaboam, Cory M., Khalil, Nasreen, Burgess, Janette K., Prêle, Cecilia M.. Wiley-Blackwell; 2018. Mitochondrial dysfunction contributes to the senescent phenotype of IPF lung fibroblasts.
Waters, David W., Blokland, Kaj E. C., Pathinayake, Prabuddha S., Burgess, Janette K., Mutsaers, Steven E., Prele, Cecilia M., Schuliga, Michael, Grainge, Christopher L., Knight, Darryl A.. American Physiological Society; 2018. Fibroblast senescence in the pathology of idiopathic pulmonary fibrosis.
Schuliga, Michael, Kanwal, Amama, Read, Jane, Blokland, Kaj E. C., Burgess, Janette K., Prele, Cecilia M., Mutsaers, Steven E., Grainge, Christopher, Thomson, Claire, James, Allen, Bartlett, Nathan W., Knight, Darryl A.. American Physiological Society; 2021. A cGAS-dependent response links DNA damage and senescence in alveolar epithelial cells: a potential drug target in IPF.
Blokland, Kaj E. C., Habibie, Habibie, Borghuis, Theo, Teitsma, Greta J., Schuliga, Michael, Melgert, Barbro N., Knight, Darryl A., Brandsma, Corry-Anke, Pouwels, Simon D., Burgess, Janette K.. MDPI AG; 2021. Regulation of cellular senescence is independent from profibrotic fibroblast-deposited ECM.
Waters, David W., Schuliga, Michael, Mutsaers, Steven E., Grainge, Christopher L., Knight, Darryl A., Pathinayake, Prabuddha S., Wei, Lan, Tan, Hui-Ying, Blokland, Kaj E. C., Jaffar, Jade, Westall, Glen P., Burgess, Janette K., Prele, Cecilia M.. MDPI AG; 2021. A senescence bystander effect in human lung fibroblasts.
Waters, David W., Blokland, Kaj E. C., Pathinayake, Prabuddha S., Wei, Lan, Schuliga, Michael, Jaffar, Jade, Westall, Glen P., Hansbro, Philip M., Prele, Cecilia M., Mutsaers, Steven E., Bartlett, Nathan W., Burgess, Janette K., Grainge, Christopher L., Knight, Darryl A.. American Thoracic Society; 2019. STAT3 regulates the onset of oxidant-induced senescence in lung fibroblasts.
Blokland, Kaj E. C., Waters, David W., Burgess, Janette K., Knight, Darryl A., Schuliga, Michael, Read, Jane, Pouwels, Simon D., Grainge, Christopher L., Jaffar, Jade, Westall, Glen, Mutsaers, Steven E., Prele, Cecilia M.. MDPI AG; 2020. Senescence of IPF lung fibroblasts disrupt alveolar epithelial cell proliferation and promote migration in wound healing.
McVey, Mark J., Maishan, Mazharul, Blokland, Kaj E. C., Bartlett, Nathan, Kuebler, Wolfgang M.. American Physiological Society; 2019. Extracellular vesicles in lung health, disease, and therapy.
Schuliga, Michael, Read, Jane, Blokland, Kaj E. C., Waters, David W., Burgess, Janette, Prele, Cecilia, Mutsaers, Steven E., Jaffar, Jade, Westall, Glen, Reid, Andrew, James, Allen, Grainge, Christopher, Knight, Darryl A.. Portland Press; 2020. Self DNA perpetuates IPF lung fibroblast senescence in a cGAS-dependent manner.
Blokland, Kaj E. C., Pouwels, Simon D., Schuliga, Michael, Knight, Darryl A., Burgess, Janette K.. Portland Press; 2020. Regulation of cellular senescence by extracellular matrix during chronic fibrotic diseases.
Blokland, Kaj E. C., Nizamoglu, Mehmet, Habibie, Habibie, Borghuis, Theo, Schuliga, Michael, Melgert, Barbro N., Knight, Darryl A., Brandsma, Corry-Anke, Pouwels, Simon D., Burgess, Janette K.. Frontiers Research Foundation; 2022. Substrate stiffness engineered to replicate disease conditions influence senescence and fibrotic responses in primary lung fibroblasts.